Crosstalk between JNK and SUMO Signaling Pathways: deSUMOylation Is Protective against H(2)O(2)-Induced Cell Injury

BACKGROUND: Oxidative stress is a key feature in the pathogenesis of several neurological disorders. Following oxidative stress stimuli a wide range of pathways are activated and contribute to cellular death. The mechanism that couples c-Jun N-terminal kinase (JNK) signaling, a key pathway in stress...

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Detalles Bibliográficos
Autores principales: Feligioni, Marco, Brambilla, Elisa, Camassa, Agata, Sclip, Alessandra, Arnaboldi, Andrea, Morelli, Federica, Antoniou, Xanthi, Borsello, Tiziana
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2011
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3229511/
https://www.ncbi.nlm.nih.gov/pubmed/22164242
http://dx.doi.org/10.1371/journal.pone.0028185

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3229511/
https://www.ncbi.nlm.nih.gov/pubmed/22164242
http://dx.doi.org/10.1371/journal.pone.0028185

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