Promoter Polymorphism G-6A, which Modulates Angiotensinogen Gene Expression, Is Associated with Non-Familial Sick Sinus Syndrome

BACKGROUND: It is well known that familial sick sinus syndrome (SSS) is caused by functional alterations of ion channels and gap junction. Limited information is available on the mechanism of age-related non-familial SSS. Although evidence shows a close link between arrhythmia and the renin-angioten...

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Detalles Bibliográficos
Autores principales: Chen, Jan-Yow, Liou, Ying-Ming, Wu, Hong-Dar Isaac, Lin, Kuo-Hung, Chang, Kuan-Cheng
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2012
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3252346/
https://www.ncbi.nlm.nih.gov/pubmed/22242192
http://dx.doi.org/10.1371/journal.pone.0029951

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3252346/
https://www.ncbi.nlm.nih.gov/pubmed/22242192
http://dx.doi.org/10.1371/journal.pone.0029951

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