Inhibition of activated NR2B gene- and caspase-3 protein-expression by glutathione following traumatic brain injury in a rat model

BACKGROUND. Traumatic brain injury (TBI) remains a major cause of death and disability. Oxidative stress is an important element of the injury cascade following TBI. Progressive compromise of antioxidant defenses and free radical-mediated lipid peroxidation are one of the major mechanisms of seconda...

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Detalles Bibliográficos
Autores principales: Arifin, Muhammad Zafrullah, Faried, Ahmad, Shahib, Muhammad Nurhalim, Wiriadisastra, Kahdar, Bisri, Tatang
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Medknow Publications & Media Pvt Ltd 2011
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3277073/
https://www.ncbi.nlm.nih.gov/pubmed/22347327
http://dx.doi.org/10.4103/1793-5482.92160

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3277073/
https://www.ncbi.nlm.nih.gov/pubmed/22347327
http://dx.doi.org/10.4103/1793-5482.92160

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