Repression of miR-142 by p300 and MAPK is required for survival signalling via gp130 during adaptive hypertrophy

An increase in cardiac workload, ultimately resulting in hypertrophy, generates oxidative stress and therefore requires the activation of both survival and growth signal pathways. Here, we wanted to characterize the regulators, targets and mechanistic roles of miR-142, a microRNA (miRNA) negatively...

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Detalles Bibliográficos
Autores principales: Sharma, Salil, Liu, Jing, Wei, Jianqin, Yuan, Huijun, Zhang, Taifang, Bishopric, Nanette H
Formato: Online Artículo Texto
Lenguaje:English
Publicado: WILEY-VCH Verlag 2012
Materias:
Research Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3407949/
https://www.ncbi.nlm.nih.gov/pubmed/22367739
http://dx.doi.org/10.1002/emmm.201200234

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3407949/
https://www.ncbi.nlm.nih.gov/pubmed/22367739
http://dx.doi.org/10.1002/emmm.201200234

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