Selenium preserves mitochondrial function, stimulates mitochondrial biogenesis, and reduces infarct volume after focal cerebral ischemia

BACKGROUND: Mitochondrial dysfunction is one of the major events responsible for activation of neuronal cell death pathways during cerebral ischemia. Trace element selenium has been shown to protect neurons in various diseases conditions. Present study is conducted to demonstrate that selenium prese...

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Detalles Bibliográficos
Autores principales: Mehta, Suresh L, Kumari, Santosh, Mendelev, Natalia, Li, P Andy
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2012
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3411431/
https://www.ncbi.nlm.nih.gov/pubmed/22776356
http://dx.doi.org/10.1186/1471-2202-13-79

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3411431/
https://www.ncbi.nlm.nih.gov/pubmed/22776356
http://dx.doi.org/10.1186/1471-2202-13-79

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