Neuroprotection by inhibiting the c-Jun N-terminal kinase pathway after cerebral ischemia occurs independently of interleukin-6 and keratinocyte-derived chemokine (KC/CXCL1) secretion

BACKGROUND: Cerebral ischemia is associated with the activation of glial cells, infiltration of leukocytes and an increase in inflammatory mediators in the ischemic brain and systemic circulation. How this inflammatory response influences lesion size and neurological outcome remains unclear. D-JNKI1...

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Detalles Bibliográficos
Autores principales: Benakis, Corinne, Vaslin, Anne, Pasquali, Christian, Hirt, Lorenz
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2012
Materias:
Short Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3416579/
https://www.ncbi.nlm.nih.gov/pubmed/22533966
http://dx.doi.org/10.1186/1742-2094-9-76

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3416579/
https://www.ncbi.nlm.nih.gov/pubmed/22533966
http://dx.doi.org/10.1186/1742-2094-9-76

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