Inhibition of Doxorubicin-Induced Senescence by PPARδ Activation Agonists in Cardiac Muscle Cells: Cooperation between PPARδ and Bcl6

Senescence and apoptosis are two distinct cellular programs that are activated in response to a variety of stresses. Low or high doses of the same stressor, i.e., the anticancer drug doxorubicin, may either induce apoptosis or senescence, respectively, in cardiac muscle cells. We have demonstrated t...

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Detalles Bibliográficos
Autores principales: Altieri, Paola, Spallarossa, Paolo, Barisione, Chiara, Garibaldi, Silvano, Garuti, Anna, Fabbi, Patrizia, Ghigliotti, Giorgio, Brunelli, Claudio
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2012
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3458009/
https://www.ncbi.nlm.nih.gov/pubmed/23049957
http://dx.doi.org/10.1371/journal.pone.0046126

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3458009/
https://www.ncbi.nlm.nih.gov/pubmed/23049957
http://dx.doi.org/10.1371/journal.pone.0046126

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