CaMKII determines mitochondrial stress responses in heart

Myocardial cell death is initiated by excessive mitochondrial Ca(2+) entry, causing Ca(2+) overload, mitochondrial permeability transition pore (mPTP) opening and dissipation of the mitochondrial inner membrane potential (ΔΨm)(1,2). However, the signaling pathways that control mitochondrial Ca(2+) e...

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Detalles Bibliográficos
Autores principales: Joiner, Mei-ling A., Koval, Olha M., Jingdong, Li, He, B. Julie, Allamargot, Chantal, Gao, Zhan, Luczak, Elizabeth D., Hall, Duane D., Fink, Brian D., Chen, Biyi, Yang, Jinying, Moore, Steven A., Scholz, Thomas D., Strack, Stefan, Mohler, Peter J., Sivitz, William I., Song, Long-Sheng, Anderson, Mark E.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: 2012
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3471377/
https://www.ncbi.nlm.nih.gov/pubmed/23051746
http://dx.doi.org/10.1038/nature11444

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3471377/
https://www.ncbi.nlm.nih.gov/pubmed/23051746
http://dx.doi.org/10.1038/nature11444

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