c-Jun NH(2)-Terminal Kinase 1/2 and Endoplasmic Reticulum Stress as Interdependent and Reciprocal Causation in Diabetic Embryopathy

Embryos exposed to high glucose exhibit aberrant maturational and cytoarchitectural cellular changes, implicating cellular organelle stress in diabetic embryopathy. c-Jun-N-terminal kinase 1/2 (JNK1/2) activation is a causal event in maternal diabetes–induced neural tube defects (NTD). However, the...

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Detalles Bibliográficos
Autores principales: Li, Xuezheng, Xu, Cheng, Yang, Peixin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Diabetes Association 2013
Materias:
Complications
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3554374/
https://www.ncbi.nlm.nih.gov/pubmed/22961085
http://dx.doi.org/10.2337/db12-0026

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3554374/
https://www.ncbi.nlm.nih.gov/pubmed/22961085
http://dx.doi.org/10.2337/db12-0026

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