Cargando…
Asymmetric N-Cadherin Expression Results in Synapse Dysfunction, Synapse Elimination, and Axon Retraction in Cultured Mouse Neurons
Synapse elimination and pruning of axon collaterals are crucial developmental events in the refinement of neuronal circuits. While a control of synapse formation by adhesion molecules is well established, the involvement of adhesion molecules in developmental synapse loss is poorly characterized. To...
Autores principales: | Pielarski, Kim N., van Stegen, Bernd, Andreyeva, Aksana, Nieweg, Katja, Jüngling, Kay, Redies, Christoph, Gottmann, Kurt |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2013
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3561303/ https://www.ncbi.nlm.nih.gov/pubmed/23382872 http://dx.doi.org/10.1371/journal.pone.0054105 |
Ejemplares similares
-
N-cadherin: stabilizing synapses
por: Arikkath, Jyothi
Publicado: (2010) -
Alzheimer's disease-related amyloid-β induces synaptotoxicity in human iPS cell-derived neurons
por: Nieweg, K, et al.
Publicado: (2015) -
Release activity-dependent control of vesicle endocytosis by the synaptic adhesion molecule N-cadherin
por: van Stegen, Bernd, et al.
Publicado: (2017) -
Synapse elimination in the developing cerebellum
por: Hashimoto, Kouichi, et al.
Publicado: (2013) -
Branch-Specific Microtubule Destabilization Mediates Axon Branch Loss during Neuromuscular Synapse Elimination
por: Brill, Monika S., et al.
Publicado: (2016)