Stat3 is a positive regulator of gap junctional intercellular communication in cultured, human lung carcinoma cells

BACKGROUND: Neoplastic transformation of cultured cells by a number of oncogenes such as src suppresses gap junctional, intercellular communication (GJIC); however, the role of Src and its effector Signal transducer and activator of transcription-3 (Stat3) upon GJIC in non small cell lung cancer (NS...

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Detalles Bibliográficos
Autores principales: Geletu, Mulu, Arulanandam, Rozanne, Greer, Samantha, Trotman-Grant, Aaron, Tomai, Evangelia, Raptis, Leda
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2012
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3575370/
https://www.ncbi.nlm.nih.gov/pubmed/23244248
http://dx.doi.org/10.1186/1471-2407-12-605

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3575370/
https://www.ncbi.nlm.nih.gov/pubmed/23244248
http://dx.doi.org/10.1186/1471-2407-12-605

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