The Influence of Flightless I on Toll-Like-Receptor-Mediated Inflammation in a Murine Model of Diabetic Wound Healing

Impaired wound healing and ulceration represent a serious complication of both type 1 and type 2 diabetes. Cytoskeletal protein Flightless I (Flii) is an important inhibitor of wound repair, and reduced Flii gene expression in fibroblasts increased migration, proliferation, and adhesion. As such it...

Descripción completa

Detalles Bibliográficos
Autores principales: Ruzehaji, Nadira, Mills, Stuart J., Melville, Elizabeth, Arkell, Ruth, Fitridge, Robert, Cowin, Allison J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2013
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3595111/
https://www.ncbi.nlm.nih.gov/pubmed/23555084
http://dx.doi.org/10.1155/2013/389792

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3595111/
https://www.ncbi.nlm.nih.gov/pubmed/23555084
http://dx.doi.org/10.1155/2013/389792

Ejemplares similares