Cargando…

Motoneuron Differentiation of Induced Pluripotent Stem Cells from SOD1G93A Mice

Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disorder mainly affecting motor neurons. Mutations in superoxide dismutase-1 (SOD-1) account for about 20% of familial ALS patients. A robust supply of motoneurons carrying the mutated gene would help understand the causes of motoneuron deat...

Descripción completa

Detalles Bibliográficos
Autores principales:	Yao, Xiao-Li, Ye, Cheng-Hui, Liu, Qiang, Wan, Jian-bo, Zhen, Jun, Xiang, Andy Peng, Li, Wei-Qiang, Wang, Yitao, Su, Huangxing, Lu, Xi-Lin
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Public Library of Science 2013
Materias:	Research Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3665598/ https://www.ncbi.nlm.nih.gov/pubmed/23724084 http://dx.doi.org/10.1371/journal.pone.0064720

Ejemplares similares

Correction: Motoneuron Differentiation of Induced Pluripotent Stem Cells from SOD1G93A Mice
por: Yao, Xiao-Li, et al.
Publicado: (2013)

Comparison of dendritic calcium transients in juvenile wild type and SOD1(G93A) mouse lumbar motoneurons
por: Quinlan, Katharina A., et al.
Publicado: (2015)

Time Course of Alterations in Adult Spinal Motoneuron Properties in the SOD1(G93A) Mouse Model of ALS
por: Huh, Seoan, et al.
Publicado: (2021)

Gene expression changes in spinal motoneurons of the SOD1(G93A) transgenic model for ALS after treatment with G-CSF
por: Henriques, Alexandre, et al.
Publicado: (2015)

Caudal–Rostral Progression of Alpha Motoneuron Degeneration in the SOD1(G93A) Mouse Model of Amyotrophic Lateral Sclerosis
por: Kirby, Alastair J., et al.
Publicado: (2022)

Simvastatin accelerated motoneurons death in SOD1(G93A) mice through inhibiting Rab7-mediated maturation of late autophagic vacuoles
por: Bai, Lin, et al.
Publicado: (2021)

Age-Related Changes in Pre- and Postsynaptic Partners of the Cholinergic C-Boutons in Wild-Type and SOD1(G93A) Lumbar Motoneurons
por: Milan, Léa, et al.
Publicado: (2015)

Erratum: Huh et al., “Time Course of Alterations in Adult Spinal Motoneuron Properties in the SOD1(G93A) Mouse Model of ALS”
Publicado: (2023)

Analytical sequence to study G-CSF effect on the transcriptome of isolated spinal motoneurons from SOD1 G93A mice, an animal model for amyotrophic lateral sclerosis
por: Henriques, Alexandre, et al.
Publicado: (2015)

Soma size and Ca(v)1.3 channel expression in vulnerable and resistant motoneuron populations of the SOD1(G93A) mouse model of ALS
por: Shoenfeld, Liza, et al.
Publicado: (2014)

Neuromuscular effects of G93A-SOD1 expression in zebrafish
por: Sakowski, Stacey A, et al.
Publicado: (2012)

Alterations in AQP4 expression and polarization in the course of motor neuron degeneration in SOD1G93A mice
por: Dai, Jiaying, et al.
Publicado: (2017)

Metabolic Changes Associated With Muscle Expression of SOD1(G93A)
por: Dobrowolny, Gabriella, et al.
Publicado: (2018)

Endothelin-1, over-expressed in SOD1(G93A) mice, aggravates injury of NSC34-hSOD1G93A cells through complicated molecular mechanism revealed by quantitative proteomics analysis
por: Zhang, Yingzhen, et al.
Publicado: (2022)

A closer look at the SOD1(G93Adl) mouse model of ALS
Publicado: (2011)

Genetic Biomarkers for ALS Disease in Transgenic SOD1(G93A) Mice
por: Calvo, Ana C., et al.
Publicado: (2012)

Characterization of somatosensory neuron involvement in the SOD1(G93A) mouse model
por: Rubio, Miguel A., et al.
Publicado: (2022)

Therapeutic rAAVrh10 Mediated SOD1 Silencing in Adult SOD1(G93A) Mice and Nonhuman Primates
por: Borel, Florie, et al.
Publicado: (2016)

Distribution Features and Potential Effects of Serotonin in the Cerebrum of SOD1 G93A Transgenic Mice
por: He, Pei, et al.
Publicado: (2022)

Adaptive Immune Neuroprotection in G93A-SOD1 Amyotrophic Lateral Sclerosis Mice
por: Banerjee, Rebecca, et al.
Publicado: (2008)

Monoaminergic control of spinal locomotor networks in SOD1(G93A) newborn mice
por: Milan, Léa, et al.
Publicado: (2014)

Histamine Is an Inducer of the Heat Shock Response in SOD1-G93A Models of ALS
por: Apolloni, Savina, et al.
Publicado: (2019)

Neuronal–glial communication perturbations in murine SOD1(G93A) spinal cord
por: MacLean, Michael, et al.
Publicado: (2022)

SIRT1 Interacts with Prepro-Orexin in the Hypothalamus in SOD1G93A Mice
por: Zhang, Gan, et al.
Publicado: (2022)

SOD1 silencing in motoneurons or glia rescues neuromuscular function in ALS mice
por: Dirren, Elisabeth, et al.
Publicado: (2015)

R13 preserves motor performance in SOD1(G93A) mice by improving mitochondrial function
por: Li, Xiao, et al.
Publicado: (2021)

Electrophysiologic Biomarkers for Assessing Disease Progression and the Effect of Riluzole in SOD1 G93A ALS Mice
por: Li, Jia, et al.
Publicado: (2013)

Postmitotic Expression of SOD1(G93A) Gene Affects the Identity of Myogenic Cells and Inhibits Myoblasts Differentiation
por: Martini, Martina, et al.
Publicado: (2015)

Muscle cells and motoneurons differentially remove mutant SOD1 causing familial amyotrophic lateral sclerosis
por: Onesto, Elisa, et al.
Publicado: (2011)

Nerve Terminal Degeneration Is Independent of Muscle Fiber Genotype in SOD1(G93A) Mice
por: Carrasco, Dario I., et al.
Publicado: (2010)

Scolopendra subspinipes mutilans attenuates neuroinflammation in symptomatic hSOD1(G93A) mice
por: Cai, MuDan, et al.
Publicado: (2013)

Neuroprotective Effect of Bexarotene in the SOD1(G93A) Mouse Model of Amyotrophic Lateral Sclerosis
por: Riancho, Javier, et al.
Publicado: (2015)

Immune response in peripheral axons delays disease progression in SOD1(G93A) mice
por: Nardo, Giovanni, et al.
Publicado: (2016)

Effects of RAGE inhibition on the progression of the disease in hSOD1(G93A) ALS mice
por: Liu, Liping, et al.
Publicado: (2020)

Repurposing of Trimetazidine for amyotrophic lateral sclerosis: A study in SOD1(G93A) mice
por: Scaricamazza, Silvia, et al.
Publicado: (2022)

Comparing therapeutic modulators of the SOD1 G93A Amyotrophic Lateral Sclerosis mouse pathophysiology
por: Lee, Albert J. B., et al.
Publicado: (2023)

Cortical glia in SOD1(G93A) mice are subtly affected by ALS-like pathology
por: Filipi, Tereza, et al.
Publicado: (2023)

Repaglinide Induces ATF6 Processing and Neuroprotection in Transgenic SOD1G93A Mice
por: Gonzalo-Gobernado, Rafael, et al.
Publicado: (2023)

Tempol Moderately Extends Survival in a hSOD1(G93A) ALS Rat Model by Inhibiting Neuronal Cell Loss, Oxidative Damage and Levels of Non-Native hSOD1(G93A) Forms
por: Linares, Edlaine, et al.
Publicado: (2013)

ALS-associated mutant SOD1(G93A )causes mitochondrial vacuolation by expansion of the intermembrane space and by involvement of SOD1 aggregation and peroxisomes
por: Higgins, Cynthia MJ, et al.
Publicado: (2003)

Cannot write session to /tmp/vufind_sessions/sess_0dpg643l4n2jlnuqqjqos3g0sh