Cargando…
SNAI1-Mediated Epithelial-Mesenchymal Transition Confers Chemoresistance and Cellular Plasticity by Regulating Genes Involved in Cell Death and Stem Cell Maintenance
Tumor cells at the tumor margin lose epithelial properties and acquire features of mesenchymal cells, a process called epithelial-to-mesenchymal transition (EMT). Recently, features of EMT were shown to be linked to cells with tumor-founding capability, so-called cancer stem cells (CSCs). Inducers o...
Autores principales: | Lim, Soyoung, Becker, Astrid, Zimmer, Andreas, Lu, Jianrong, Buettner, Reinhard, Kirfel, Jutta |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2013
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3684605/ https://www.ncbi.nlm.nih.gov/pubmed/23799116 http://dx.doi.org/10.1371/journal.pone.0066558 |
Ejemplares similares
-
Loss of SNAI1 induces cellular plasticity in invasive triple-negative breast cancer cells
por: Tsirigoti, Chrysoula, et al.
Publicado: (2022) -
MiR-203 downregulation is responsible for chemoresistance in human glioblastoma by promoting epithelial-mesenchymal transition via SNAI2
por: Liao, Hongzhan, et al.
Publicado: (2015) -
SNAI1 recruits HDAC1 to suppress SNAI2 transcription during epithelial to mesenchymal transition
por: Sundararajan, Vignesh, et al.
Publicado: (2019) -
miR302 regulates SNAI1 expression to control mesangial cell plasticity
por: De Chiara, L., et al.
Publicado: (2017) -
Corrigendum: miR302 regulates SNAI1 expression to control mesangial cell plasticity
por: De Chiara, Letizia, et al.
Publicado: (2017)