Yersinia pestis Subverts the Dermal Neutrophil Response in a Mouse Model of Bubonic Plague

The majority of human Yersinia pestis infections result from introduction of bacteria into the skin by the bite of an infected flea. Once in the dermis, Y. pestis can evade the host’s innate immune response and subsequently disseminate to the draining lymph node (dLN). There, the pathogen replicates...

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Detalles Bibliográficos
Autores principales: Shannon, Jeffrey G., Hasenkrug, Aaron M., Dorward, David W., Nair, Vinod, Carmody, Aaron B., Hinnebusch, B. Joseph
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Society of Microbiology 2013
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3760243/
https://www.ncbi.nlm.nih.gov/pubmed/23982068
http://dx.doi.org/10.1128/mBio.00170-13

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3760243/
https://www.ncbi.nlm.nih.gov/pubmed/23982068
http://dx.doi.org/10.1128/mBio.00170-13

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