Ethanol Metabolism Modifies Hepatic Protein Acylation in Mice

Mitochondrial protein acetylation increases in response to chronic ethanol ingestion in mice, and is thought to reduce mitochondrial function and contribute to the pathogenesis of alcoholic liver disease. The mitochondrial deacetylase SIRT3 regulates the acetylation status of several mitochondrial p...

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Detalles Bibliográficos
Autores principales: Fritz, Kristofer S., Green, Michelle F., Petersen, Dennis R., Hirschey, Matthew D.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2013
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3779192/
https://www.ncbi.nlm.nih.gov/pubmed/24073283
http://dx.doi.org/10.1371/journal.pone.0075868

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3779192/
https://www.ncbi.nlm.nih.gov/pubmed/24073283
http://dx.doi.org/10.1371/journal.pone.0075868

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