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Alzheimer's disease-associated peptide Aβ(42) mobilizes ER Ca(2+) via InsP(3)R-dependent and -independent mechanisms
Dysregulation of Ca(2+) homeostasis is considered to contribute to the toxic action of the Alzheimer's disease (AD)-associated amyloid-β-peptide (Aβ). Ca(2+) fluxes across the plasma membrane and release from intracellular stores have both been reported to underlie the Ca(2+) fluxes induced by...
| Autores principales: | , , , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Frontiers Media S.A.
2013
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3817845/ https://www.ncbi.nlm.nih.gov/pubmed/24204331 http://dx.doi.org/10.3389/fnmol.2013.00036 |