A Cdx4-Sall4 Regulatory Module Controls the Transition from Mesoderm Formation to Embryonic Hematopoiesis

Deletion of caudal/cdx genes alters hox gene expression and causes defects in posterior tissues and hematopoiesis. Yet, the defects in hox gene expression only partially explain these phenotypes. To gain deeper insight into Cdx4 function, we performed chromatin immunoprecipitation sequencing (ChIP-s...

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Detalles Bibliográficos
Autores principales: Paik, Elizabeth J., Mahony, Shaun, White, Richard M., Price, Emily N., DiBiase, Anthony, Dorjsuren, Bilguujin, Mosimann, Christian, Davidson, Alan J., Gifford, David, Zon, Leonard I.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2013
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3841246/
https://www.ncbi.nlm.nih.gov/pubmed/24286030
http://dx.doi.org/10.1016/j.stemcr.2013.10.001

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3841246/
https://www.ncbi.nlm.nih.gov/pubmed/24286030
http://dx.doi.org/10.1016/j.stemcr.2013.10.001

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