Strand invasion by HLTF as a mechanism for template switch in fork rescue

Stalling of replication forks at unrepaired DNA lesions can result in discontinuities opposite the damage in the newly synthesized DNA strand. Translesion synthesis or facilitating the copy from the newly synthesized strand of the sister duplex by template switching can overcome such discontinuities...

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Detalles Bibliográficos
Autores principales: Burkovics, Peter, Sebesta, Marek, Balogh, David, Haracska, Lajos, Krejci, Lumir
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Oxford University Press 2014
Materias:
Genome Integrity, Repair and Replication
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3919600/
https://www.ncbi.nlm.nih.gov/pubmed/24198246
http://dx.doi.org/10.1093/nar/gkt1040

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3919600/
https://www.ncbi.nlm.nih.gov/pubmed/24198246
http://dx.doi.org/10.1093/nar/gkt1040

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