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Non-canonical p53 signaling to promote invasion
It has been known for a number of years that mutated “inactive” p53 proteins still capable of binding to DNA per se, can bind to DNA sequences that are non-canonical for p53, with for example, a resultant increase in the transcription and expression of growth factor receptors such as ERBB1,(1)(,)(2)...
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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Landes Bioscience
2013
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3926882/ https://www.ncbi.nlm.nih.gov/pubmed/24025254 http://dx.doi.org/10.4161/cbt.26174 |