Nitric Oxide Synthase Inhibition Attenuates Cardiac Response to Hemodilution with Viscogenic Plasma Expander

BACKGROUND AND OBJECTIVES: Increased vascular wall shear stress by elevated plasma viscosity significantly enhances the endothelial nitric oxide synthase (eNOS) activity during an acute isovolemic hemodilution. Also the modulation of plasma viscosity has effects on the cardiac function that were rev...

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Detalles Bibliográficos
Autores principales: Chatpun, Surapong, Cabrales, Pedro
Formato: Online Artículo Texto
Lenguaje:English
Publicado: The Korean Society of Cardiology 2014
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3958604/
https://www.ncbi.nlm.nih.gov/pubmed/24653740
http://dx.doi.org/10.4070/kcj.2014.44.2.105

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3958604/
https://www.ncbi.nlm.nih.gov/pubmed/24653740
http://dx.doi.org/10.4070/kcj.2014.44.2.105

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