Acute nitric oxide synthase inhibition and endothelin-1-dependent arterial pressure elevation

Key evidence that endogenous nitric oxide (NO) inhibits the continuous, endothelin (ET)-1-mediated drive to elevate arterial pressure includes demonstrations that ET-1 mediates a significant component of the pressure elevated by acute exposure to NO synthase (NOS) inhibitors. This review examines th...

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Detalles Bibliográficos
Autor principal: Rapoport, Robert M.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2014
Materias:
Pharmacology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3978292/
https://www.ncbi.nlm.nih.gov/pubmed/24744731
http://dx.doi.org/10.3389/fphar.2014.00057

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3978292/
https://www.ncbi.nlm.nih.gov/pubmed/24744731
http://dx.doi.org/10.3389/fphar.2014.00057

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