Erk5 is a mediator to TGFβ1-induced loss of phenotype and function in human podocytes

Background: Podocytes are highly specialized cells integral to the normal functioning kidney, however, in diabetic nephropathy injury occurs leading to a compromised phenotype and podocyte dysfunction which critically produces podocyte loss with subsequent renal impairment. TGFβ1 holds a major role...

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Detalles Bibliográficos
Autores principales: Badshah, Irbaz I., Baines, Deborah L., Dockrell, Mark E.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2014
Materias:
Pharmacology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4001011/
https://www.ncbi.nlm.nih.gov/pubmed/24795631
http://dx.doi.org/10.3389/fphar.2014.00071

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4001011/
https://www.ncbi.nlm.nih.gov/pubmed/24795631
http://dx.doi.org/10.3389/fphar.2014.00071

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