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JNK interaction with Sab mediates ER stress induced inhibition of mitochondrial respiration and cell death
Our aim was to better understand the mechanism and importance of sustained c-Jun N-terminal kinase (JNK) activation in endoplasmic reticulum (ER) stress and effects of ER stress on mitochondria by determining the role of mitochondrial JNK binding protein, Sab. Tunicamycin or brefeldin A induced a ra...
Autores principales: | Win, S, Than, T A, Fernandez-Checa, J C, Kaplowitz, N |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group
2014
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4040675/ https://www.ncbi.nlm.nih.gov/pubmed/24407242 http://dx.doi.org/10.1038/cddis.2013.522 |
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