Overexpression of BID in thyroids of transgenic mice increases sensitivity to iodine-induced autoimmune thyroiditis

BACKGROUND: BID functions as a bridge molecule between death-receptor and mitochondrial related apoptotic pathways to amplify apoptotic signaling. Our previous studies have demonstrated a substantial increase in BID expression in primary normal thyroid epithelia cells treated with inflammatory cytok...

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Detalles Bibliográficos
Autores principales: Wang, Su He, Fan, Yongyi, Baker, James R
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2014
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4083877/
https://www.ncbi.nlm.nih.gov/pubmed/24957380
http://dx.doi.org/10.1186/1479-5876-12-180

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4083877/
https://www.ncbi.nlm.nih.gov/pubmed/24957380
http://dx.doi.org/10.1186/1479-5876-12-180

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