Overexpression of Shox2 Leads to Congenital Dysplasia of the Temporomandibular Joint in Mice

Our previous study reported that inactivation of Shox2 led to dysplasia and ankylosis of the temporomandibular joint (TMJ), and that replacing Shox2 with human Shox partially rescued the phenotype with a prematurely worn out articular disc. However, the mechanisms of Shox2 activity in TMJ developmen...

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Detalles Bibliográficos
Autores principales: Li, Xihai, Liang, Wenna, Ye, Hongzhi, Weng, Xiaping, Liu, Fayuan, Liu, Xianxiang
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2014
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4159784/
https://www.ncbi.nlm.nih.gov/pubmed/25062348
http://dx.doi.org/10.3390/ijms150813135

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4159784/
https://www.ncbi.nlm.nih.gov/pubmed/25062348
http://dx.doi.org/10.3390/ijms150813135

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