Tissue-specific deletion of Crry from mouse proximal tubular epithelial cells increases susceptibility to renal ischemia reperfusion injury

The murine cell surface protein Crry is a key complement regulator with similar activities to human membrane cofactor protein (MCP) and decay-accelerating factor. MCP plays a critical role in preventing complement-mediated tissue injury and its mutation has been implicated in several human kidney di...

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Detalles Bibliográficos
Autores principales: Miao, Jing, Lesher, Allison M., Miwa, Takashi, Sato, Sayaka, Gullipalli, Damodar, Song, Wen-Chao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: 2014
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4182132/
https://www.ncbi.nlm.nih.gov/pubmed/24850152
http://dx.doi.org/10.1038/ki.2014.103

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4182132/
https://www.ncbi.nlm.nih.gov/pubmed/24850152
http://dx.doi.org/10.1038/ki.2014.103

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