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Tissue-specific deletion of Crry from mouse proximal tubular epithelial cells increases susceptibility to renal ischemia reperfusion injury

The murine cell surface protein Crry is a key complement regulator with similar activities to human membrane cofactor protein (MCP) and decay-accelerating factor. MCP plays a critical role in preventing complement-mediated tissue injury and its mutation has been implicated in several human kidney di...

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Detalles Bibliográficos
Autores principales:	Miao, Jing, Lesher, Allison M., Miwa, Takashi, Sato, Sayaka, Gullipalli, Damodar, Song, Wen-Chao
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	2014
Materias:	Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4182132/ https://www.ncbi.nlm.nih.gov/pubmed/24850152 http://dx.doi.org/10.1038/ki.2014.103

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