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Receptor interacting protein 140 attenuates endoplasmic reticulum stress in neurons and protects against cell death
Inositol 1, 4, 5-trisphosphate receptor (IP(3)R)-mediated Ca(2+) release from the endoplasmic reticulum (ER) triggers many physiological responses in neurons and when uncontrolled can cause ER stress that contributes to neurological disease. Here we show that the unfolded protein response (UPR) in n...
Autores principales: | , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
2014
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4200015/ https://www.ncbi.nlm.nih.gov/pubmed/25066731 http://dx.doi.org/10.1038/ncomms5487 |