The elimination of miR-23a in heat-stressed cells promotes NOXA-induced cell death and is prevented by HSP70

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Protein-damaging stress stimulates cell destruction through apoptosis; however, non-lethal proteotoxic stress induces an adaptive response leading to the increased synthesis of heat shock proteins, which inhibit apoptosis. In this study, we sought to determine the mechanism responsible for the accum...

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Detalles Bibliográficos
Autores principales: Roufayel, R, Johnston, D S, Mosser, D D
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2014
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4260742/
https://www.ncbi.nlm.nih.gov/pubmed/25429623
http://dx.doi.org/10.1038/cddis.2014.484

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4260742/
https://www.ncbi.nlm.nih.gov/pubmed/25429623
http://dx.doi.org/10.1038/cddis.2014.484

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