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Tau pathogenesis is promoted by Aβ1-42 but not Aβ1-40
BACKGROUND: The relationship between the pathogenic amyloid β-peptide species Aβ1–42 and tau pathology has been well studied and suggests that Aβ1–42 can accelerate tau pathology in vitro and in vivo. The manners if any in which Aβ1–40 interacts with tau remains poorly understood. In order to answer...
| Autores principales: | , , , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
BioMed Central
2014
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4277831/ https://www.ncbi.nlm.nih.gov/pubmed/25417177 http://dx.doi.org/10.1186/1750-1326-9-52 |