Cargando…
Distinct Cellular Calcium Metabolism in Radiation-sensitive RKO Human Colorectal Cancer Cells
Radiation therapy for variety of human solid tumors utilizes mechanism of cell death after DNA damage caused by radiation. In response to DNA damage, cytochrome c was released from mitochondria by activation of pro-apoptotic Bcl-2 family proteins, and then elicits massive Ca(2+) release from the ER...
Autores principales: | Kim, Yun Tai, Jo, Soo Shin, Park, Young Jun, Lee, Myung Za, Suh, Chang Kook |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
The Korean Physiological Society and The Korean Society of Pharmacology
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4296041/ https://www.ncbi.nlm.nih.gov/pubmed/25598666 http://dx.doi.org/10.4196/kjpp.2014.18.6.509 |
Ejemplares similares
-
The RKO story /
por: Jewell, Richard B. -
Universal response in the RKO colon cancer cell line to distinct antimitotic therapies
por: Lorz, Alexander, et al.
Publicado: (2018) -
Ursolic acid induces apoptosis and anoikis in colorectal carcinoma RKO cells
por: Zheng, Jia-Lu, et al.
Publicado: (2021) -
Knockdown of SAR1B suppresses proliferation and induces apoptosis of RKO colorectal cancer cells
por: Lu, Yong, et al.
Publicado: (2020) -
An iron-dependent metabolic vulnerability underlies VPS34-dependence in RKO cancer cells
por: Kobylarz, Marek J., et al.
Publicado: (2020)