Arrhythmogenic Calmodulin Mutations Disrupt Intracellular Cardiomyocyte Ca(2+) Regulation by Distinct Mechanisms

BACKGROUND: Calmodulin (CaM) mutations have been identified recently in subjects with congenital long QT syndrome (LQTS) or catecholaminergic polymorphic ventricular tachycardia (CPVT), but the mechanisms responsible for these divergent arrhythmia‐susceptibility syndromes in this context are unknown...

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Detalles Bibliográficos
Autores principales: Yin, Guo, Hassan, Faisal, Haroun, Ayman R., Murphy, Lisa L., Crotti, Lia, Schwartz, Peter J., George, Alfred L., Satin, Jonathan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Blackwell Publishing Ltd 2014
Materias:
Original Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4309107/
https://www.ncbi.nlm.nih.gov/pubmed/24958779
http://dx.doi.org/10.1161/JAHA.114.000996

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4309107/
https://www.ncbi.nlm.nih.gov/pubmed/24958779
http://dx.doi.org/10.1161/JAHA.114.000996

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