Segregation of a Spontaneous Klrd1 (CD94) Mutation in DBA/2 Mouse Substrains

Current model DBA/2J (D2J) mice lack CD94 expression due to a deletion spanning the last coding exon of the Klrd1 gene that occurred in the mid- to late 1980s. In contrast, DBA/2JRj (D2Rj) mice, crosses derived from DBA/2J before 1984, and C57BL/6J (B6) mice lack the deletion and have normal CD94 ex...

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Detalles Bibliográficos
Autores principales: Shin, Dai-Lun, Pandey, Ashutosh K., Ziebarth, Jesse Dylan, Mulligan, Megan K., Williams, Robert W., Geffers, Robert, Hatesuer, Bastian, Schughart, Klaus, Wilk, Esther
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Genetics Society of America 2014
Materias:
Investigations
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4321031/
https://www.ncbi.nlm.nih.gov/pubmed/25520036
http://dx.doi.org/10.1534/g3.114.015164

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4321031/
https://www.ncbi.nlm.nih.gov/pubmed/25520036
http://dx.doi.org/10.1534/g3.114.015164

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