Silencing collapsin response mediator protein-2 reprograms macrophage phenotype and improves infarct healing in experimental myocardial infarction model

BACKGROUND: Delayed M1 toward M2 macrophage phenotype transition is considered one of the major causes for the impaired healing after myocardial infarction (MI). While searching for molecules that modulate M1 and M2 macrophage polarization, we identified collapsin response mediator protein-2 (CRMP2)...

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Detalles Bibliográficos
Autores principales: Zhou, Long-Shu, Zhao, Guo-Long, Liu, Qiang, Jiang, Shu-Cai, Wang, Yun, Zhang, Dong-Mei
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2015
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4328069/
https://www.ncbi.nlm.nih.gov/pubmed/25685072
http://dx.doi.org/10.1186/s12950-015-0053-8

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4328069/
https://www.ncbi.nlm.nih.gov/pubmed/25685072
http://dx.doi.org/10.1186/s12950-015-0053-8

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