Differential effects of buffer pH on Ca(2+)-induced ROS emission with inhibited mitochondrial complexes I and III

Excessive mitochondrial reactive oxygen species (ROS) emission is a critical component in the etiology of ischemic injury. Complex I and complex III of the electron transport chain are considered the primary sources of ROS emission during cardiac ischemia and reperfusion (IR) injury. Several factors...

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Detalles Bibliográficos
Autores principales: Lindsay, Daniel P., Camara, Amadou K. S., Stowe, David F., Lubbe, Ryan, Aldakkak, Mohammed
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2015
Materias:
Physiology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4354303/
https://www.ncbi.nlm.nih.gov/pubmed/25805998
http://dx.doi.org/10.3389/fphys.2015.00058

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4354303/
https://www.ncbi.nlm.nih.gov/pubmed/25805998
http://dx.doi.org/10.3389/fphys.2015.00058

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