Lack of parvalbumin in mice leads to behavioral deficits relevant to all human autism core symptoms and related neural morphofunctional abnormalities

Gene mutations and gene copy number variants are associated with autism spectrum disorders (ASDs). Affected gene products are often part of signaling networks implicated in synapse formation and/or function leading to alterations in the excitation/inhibition (E/I) balance. Although the network of pa...

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Detalles Bibliográficos
Autores principales: Wöhr, M, Orduz, D, Gregory, P, Moreno, H, Khan, U, Vörckel, K J, Wolfer, D P, Welzl, H, Gall, D, Schiffmann, S N, Schwaller, B
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2015
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4354349/
https://www.ncbi.nlm.nih.gov/pubmed/25756808
http://dx.doi.org/10.1038/tp.2015.19

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4354349/
https://www.ncbi.nlm.nih.gov/pubmed/25756808
http://dx.doi.org/10.1038/tp.2015.19

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