Cargando…

Poly (I:C) therapy decreases cerebral ischaemia/reperfusion injury viaTLR3-mediated prevention of Fas/FADD interaction

Toll-like receptor (TLR)-mediated signalling plays a role in cerebral ischaemia/reperfusion (I/R) injury. Modulation of TLRs has been reported to protect against cerebral I/R injury. This study examined whether modulation of TLR3 with poly (I:C) will induce protection against cerebral I/R injury. Mi...

Descripción completa

Detalles Bibliográficos
Autores principales:	Zhang, Xia, Ha, Tuanzhu, Lu, Chen, Lam, Fred, Liu, Li, Schweitzer, John, Kalbfleisch, John, Kao, Race L, Williams, David L, Li, Chuanfu
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	BlackWell Publishing Ltd 2015
Materias:	Original Articles
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4369813/ https://www.ncbi.nlm.nih.gov/pubmed/25351293 http://dx.doi.org/10.1111/jcmm.12456

Ejemplares similares

TLR3 Mediates Repair and Regeneration of Damaged Neonatal Heart through Glycolysis Dependent YAP1 Regulated miR-152 Expression
por: Wang, Xiaohui, et al.
Publicado: (2018)

MicroRNA-214 protects against hypoxia/reoxygenation induced cell damage and myocardial ischemia/reperfusion injury via suppression of PTEN and Bim1 expression
por: Wang, Xiaohui, et al.
Publicado: (2016)

The TLR9 Ligand, CpG‐ODN, Induces Protection against Cerebral Ischemia/Reperfusion Injury via Activation of PI3K/Akt Signaling
por: Lu, Chen, et al.
Publicado: (2014)

Fist/Hipk3: A FAS/Fadd-Interacting Serine/Threonine Kinase That Induces Fadd Phosphorylation and Inhibits FAS-Mediated Jun Nh(2)-Terminal Kinase Activation
por: Rochat-Steiner, Véronique, et al.
Publicado: (2000)

The Fas/FADD death domain complex structure unravels signaling by receptor clustering
por: Scott, Fiona L., et al.
Publicado: (2008)

Activation of Myocardial Phosphoinositide-3-Kinase p110α Ameliorates Cardiac Dysfunction and Improves Survival in Polymicrobial Sepsis
por: Li, Chuanfu, et al.
Publicado: (2012)

Super-Resolution Imaging of Fas/CD95 Reorganization Induced by Membrane-Bound Fas Ligand Reveals Nanoscale Clustering Upstream of FADD Recruitment
por: Frazzette, Nicholas, et al.
Publicado: (2022)

The Fas–FADD death domain complex structure reveals the basis of DISC assembly and disease mutations
por: Wang, Liwei, et al.
Publicado: (2010)

AFP promotes HCC progression by suppressing the HuR-mediated Fas/FADD apoptotic pathway
por: Chen, Tianke, et al.
Publicado: (2020)

Inhibition of Fas-Associated Death Domain-Containing Protein (FADD) Protects against Myocardial Ischemia/Reperfusion Injury in a Heart Failure Mouse Model
por: Fan, Qian, et al.
Publicado: (2013)

Scavenger Receptor Class A Plays a Central Role in Mediating Mortality and the Development of the Pro-Inflammatory Phenotype in Polymicrobial Sepsis
por: Ozment, Tammy R., et al.
Publicado: (2012)

NFκB activation by Fas is mediated through FADD, caspase-8, and RIP and is inhibited by FLIP
por: Kreuz, Sebastian, et al.
Publicado: (2004)

Novel Compound Heterozygote Variations in FADD Identified to Cause FAS-Associated Protein with Death Domain Deficiency
por: Kohn, Lisa A., et al.
Publicado: (2020)

E1B 19K Inhibits Fas-mediated Apoptosis through FADD-dependent Sequestration of FLICE
por: Perez, Denise, et al.
Publicado: (1998)

Engagement of Fas on Macrophages Modulates Poly I:C Induced Cytokine Production with Specific Enhancement of IP-10
por: Lyons, Caitriona, et al.
Publicado: (2015)

Knock down of Fas-Associated Protein with Death Domain (FADD) Sensitizes Osteosarcoma to TNFα-induced Cell Death
por: Hollomon, Mario G., et al.
Publicado: (2020)

TLR3 Agonist Poly-IC Induces IL-33 and Promotes Myelin Repair
por: Natarajan, Chandramohan, et al.
Publicado: (2016)

The Response of Tissue Mast Cells to TLR3 Ligand Poly(I:C) Treatment
por: Witczak, Piotr, et al.
Publicado: (2020)

TLR3 agonists: RGC100, ARNAX, and poly-IC: a comparative review
por: Komal, Asma, et al.
Publicado: (2021)

FADD adaptor in cancer
por: Tourneur, Léa, et al.
Publicado: (2005)

Complement in ischaemia–reperfusion injury and transplantation
por: Howard, Mark C., et al.
Publicado: (2021)

Ionizing Radiation and Chemotherapeutic Drugs Induce Apoptosis in Lymphocytes in the Absence of FAS or Fadd/Mort1 Signaling: Implications for Cancer Therapy
por: Newton, Kim, et al.
Publicado: (2000)

Analysis of methylation and mRNA expression status ofFADD andFAS genes in patients with oral squamous cell carcinoma
por: Saberi, Eshaghali, et al.
Publicado: (2014)

Endothelial HSPA12B Exerts Protection Against Sepsis-Induced Severe Cardiomyopathy via Suppression of Adhesion Molecule Expression by miR-126
por: Zhang, Xia, et al.
Publicado: (2020)

Poly(I:C) preconditioning protects the heart against myocardial ischemia/reperfusion injury through TLR3/PI3K/Akt-dependent pathway
por: Chen, Erya, et al.
Publicado: (2020)

Poly(I:C)-Induced Mesenchymal Stem Cells Protect the Kidney Against Ischemia/Reperfusion Injury via the TLR3/PI3K Pathway
por: Chen, Tian, et al.
Publicado: (2021)

Correction to: Complement in ischaemia–reperfusion injury and transplantation
por: Howard, Mark C., et al.
Publicado: (2022)

Mitochondrial mechanisms and therapeutics in ischaemia reperfusion injury
por: Martin, Jack L., et al.
Publicado: (2018)

Necroptosis in Hepatosteatotic Ischaemia-Reperfusion Injury
por: Baidya, Raji, et al.
Publicado: (2020)

The role of dendritic cells regulated by HMGB1/TLR4 signalling pathway in myocardial ischaemia reperfusion injury
por: Xue, Jiyang, et al.
Publicado: (2019)

Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathway
por: Jiang, Qun, et al.
Publicado: (2008)

MiR-7a is an important mediator in Fas-associated protein with death domain (FADD)-regulated expression of focal adhesion kinase (FAK)
por: Liu, Yingting, et al.
Publicado: (2016)

Crocetin exploits p53-induced death domain (PIDD) and FAS-associated death domain (FADD) proteins to induce apoptosis in colorectal cancer
por: Ray, Pallab, et al.
Publicado: (2016)

Distinct and complementary functions of MDA5 and TLR3 in poly(I:C)-mediated activation of mouse NK cells
por: McCartney, Stephen, et al.
Publicado: (2009)

Isoflurane Attenuates Cerebral Ischaemia–Reperfusion Injury via the TLR4-NLRP3 Signalling Pathway in Diabetic Mice
por: Zhang, Ya-Jun, et al.
Publicado: (2022)

FADD the bad in head and neck cancer
por: Dent, Paul
Publicado: (2013)

Long-term activation of TLR3 by Poly(I:C) induces inflammation and impairs lung function in mice
por: Stowell, Nicole C, et al.
Publicado: (2009)

A new model for evaluation of thrombosis and ischaemia/reperfusion injury
por: Ravn, HB, et al.
Publicado: (1998)

Poly (ADP‐ribose) polymerase inhibition protects against myocardial ischaemia/reperfusion injury via suppressing mitophagy
por: Cao, Shengchuan, et al.
Publicado: (2019)

l‐Fucose prevention of renal ischaemia/reperfusion injury in Mice
por: Howard, Mark C., et al.
Publicado: (2019)

Cannot write session to /tmp/vufind_sessions/sess_g2qtq1974pm57v876co6ka8jgv