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Pharmacological blockade of the fatty acid amide hydrolase (FAAH) alters neural proliferation, apoptosis and gliosis in the rat hippocampus, hypothalamus and striatum in a negative energy context

Endocannabinoids participate in the control of neurogenesis, neural cell death and gliosis. The pharmacological effect of the fatty acid amide hydrolase (FAAH) inhibitor URB597, which limits the endocannabinoid degradation, was investigated in the present study. Cell proliferation (phospho-H3(+) or...

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Detalles Bibliográficos
Autores principales: Rivera, Patricia, Bindila, Laura, Pastor, Antoni, Pérez-Martín, Margarita, Pavón, Francisco J., Serrano, Antonia, de la Torre, Rafael, Lutz, Beat, Rodríguez de Fonseca, Fernando, Suárez, Juan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4375993/
https://www.ncbi.nlm.nih.gov/pubmed/25870539
http://dx.doi.org/10.3389/fncel.2015.00098