Circulating NOS3 Modulates Left Ventricular Remodeling following Reperfused Myocardial Infarction

PURPOSE: Nitric oxide (NO) is constitutively produced and released from the endothelium and several blood cell types by the isoform 3 of the NO synthase (NOS3). We have shown that NO protects against myocardial ischemia/reperfusion (I/R) injury and that depletion of circulating NOS3 increases within...

Descripción completa

Detalles Bibliográficos
Autores principales: Gorressen, Simone, Stern, Manuel, van de Sandt, Annette M., Cortese-Krott, Miriam M., Ohlig, Jan, Rassaf, Tienush, Gödecke, Axel, Fischer, Jens W., Heusch, Gerd, Merx, Marc W., Kelm, Malte
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2015
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4397096/
https://www.ncbi.nlm.nih.gov/pubmed/25875863
http://dx.doi.org/10.1371/journal.pone.0120961

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4397096/
https://www.ncbi.nlm.nih.gov/pubmed/25875863
http://dx.doi.org/10.1371/journal.pone.0120961

Ejemplares similares