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Circulating NOS3 Modulates Left Ventricular Remodeling following Reperfused Myocardial Infarction

PURPOSE: Nitric oxide (NO) is constitutively produced and released from the endothelium and several blood cell types by the isoform 3 of the NO synthase (NOS3). We have shown that NO protects against myocardial ischemia/reperfusion (I/R) injury and that depletion of circulating NOS3 increases within...

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Detalles Bibliográficos
Autores principales:	Gorressen, Simone, Stern, Manuel, van de Sandt, Annette M., Cortese-Krott, Miriam M., Ohlig, Jan, Rassaf, Tienush, Gödecke, Axel, Fischer, Jens W., Heusch, Gerd, Merx, Marc W., Kelm, Malte
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Public Library of Science 2015
Materias:	Research Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4397096/ https://www.ncbi.nlm.nih.gov/pubmed/25875863 http://dx.doi.org/10.1371/journal.pone.0120961

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