The Role of Excitotoxic Programmed Necrosis in Acute Brain Injury

Excitotoxicity involves the excessive release of glutamate from presynaptic nerve terminals and from reversal of astrocytic glutamate uptake, when there is excessive neuronal depolarization. N-methyl-d-aspartate (NMDA) receptors, a subtype of glutamate receptor, are activated in postsynaptic neurons...

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Detalles Bibliográficos
Autor principal: Fujikawa, Denson G.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Research Network of Computational and Structural Biotechnology 2015
Materias:
Mini Review
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4398818/
https://www.ncbi.nlm.nih.gov/pubmed/25893083
http://dx.doi.org/10.1016/j.csbj.2015.03.004

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4398818/
https://www.ncbi.nlm.nih.gov/pubmed/25893083
http://dx.doi.org/10.1016/j.csbj.2015.03.004

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