Variation in complement protein C1q is not a major contributor to cognitive impairment in Parkinson’s disease

Traditional dogma regarding the brain as an immune exempt organ has changed in recent years. New research has highlighted the role of the classical complement cascade in both synaptic elimination and function, driven largely by the role of the pathway initiating protein C1q. Given the links between...

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Detalles Bibliográficos
Autores principales: Carbutt, Sophia, Duff, Jennifer, Yarnall, Alison, Burn, David J., Hudson, Gavin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier Scientific Publishers Ireland 2015
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4407898/
https://www.ncbi.nlm.nih.gov/pubmed/25817358
http://dx.doi.org/10.1016/j.neulet.2015.03.048

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4407898/
https://www.ncbi.nlm.nih.gov/pubmed/25817358
http://dx.doi.org/10.1016/j.neulet.2015.03.048

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