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Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds

BACKGROUND: Retinoic acid receptor-related orphan receptor gamma t (RORγt) is the master regulator of Th17 cell differentiation, which plays a critical role in the pathology of several autoimmune diseases. By directing Th17 cells function, RORγt could be a potential target for drug development for T...

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Autores principales: Ding, Qingfeng, Zhao, Mei, Bai, Chuan, Yu, Bolan, Huang, Zhaofeng
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4446823/
https://www.ncbi.nlm.nih.gov/pubmed/26021566
http://dx.doi.org/10.1186/s12865-015-0097-9
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author Ding, Qingfeng
Zhao, Mei
Bai, Chuan
Yu, Bolan
Huang, Zhaofeng
author_facet Ding, Qingfeng
Zhao, Mei
Bai, Chuan
Yu, Bolan
Huang, Zhaofeng
author_sort Ding, Qingfeng
collection PubMed
description BACKGROUND: Retinoic acid receptor-related orphan receptor gamma t (RORγt) is the master regulator of Th17 cell differentiation, which plays a critical role in the pathology of several autoimmune diseases. By directing Th17 cells function, RORγt could be a potential target for drug development for Th17 related autoimmune disease. METHODS: A Jurkat cell-based reporter assay system was used for screening RORγt inhibitors from a drug-like chemical library, following with mouse Th17 cells differentiation study to identify the effect of targeted compounds in primary T cells. 293T cell-based reporter assay was conducted to determine the cell specificity, and MTT assay was performed to determine the cell toxicity of those compounds. RESULTS: In this study, we identified four lead compounds that suppressed RORγt activity, Th17 differentiation and IL-17A secretion. These candidates displayed inhibition ability on RORγt activity in T cell derived Jurkat cell, but not in 293 T cell, which indicated the restricted effects of these compounds to other cells or tissues. Futhermore, our results demonstrated that these candidates exhibited more robust inhibitory on IL-17 F transcription expression than IL-17A, which is different from one reported compound, SR1001, that mainly suppressed IL-17A, rather than IL-17 F production. CONCLUSIONS: Our study discovered four novel compounds that inhibited RORγt activity and Th17 function, which indicates their potential in therapeutic application of Th17 related autoimmune disorders.
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spelling pubmed-44468232015-05-29 Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds Ding, Qingfeng Zhao, Mei Bai, Chuan Yu, Bolan Huang, Zhaofeng BMC Immunol Research BACKGROUND: Retinoic acid receptor-related orphan receptor gamma t (RORγt) is the master regulator of Th17 cell differentiation, which plays a critical role in the pathology of several autoimmune diseases. By directing Th17 cells function, RORγt could be a potential target for drug development for Th17 related autoimmune disease. METHODS: A Jurkat cell-based reporter assay system was used for screening RORγt inhibitors from a drug-like chemical library, following with mouse Th17 cells differentiation study to identify the effect of targeted compounds in primary T cells. 293T cell-based reporter assay was conducted to determine the cell specificity, and MTT assay was performed to determine the cell toxicity of those compounds. RESULTS: In this study, we identified four lead compounds that suppressed RORγt activity, Th17 differentiation and IL-17A secretion. These candidates displayed inhibition ability on RORγt activity in T cell derived Jurkat cell, but not in 293 T cell, which indicated the restricted effects of these compounds to other cells or tissues. Futhermore, our results demonstrated that these candidates exhibited more robust inhibitory on IL-17 F transcription expression than IL-17A, which is different from one reported compound, SR1001, that mainly suppressed IL-17A, rather than IL-17 F production. CONCLUSIONS: Our study discovered four novel compounds that inhibited RORγt activity and Th17 function, which indicates their potential in therapeutic application of Th17 related autoimmune disorders. BioMed Central 2015-05-29 /pmc/articles/PMC4446823/ /pubmed/26021566 http://dx.doi.org/10.1186/s12865-015-0097-9 Text en © Ding et al.; licensee BioMed Central. 2015 This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.
spellingShingle Research
Ding, Qingfeng
Zhao, Mei
Bai, Chuan
Yu, Bolan
Huang, Zhaofeng
Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds
title Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds
title_full Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds
title_fullStr Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds
title_full_unstemmed Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds
title_short Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds
title_sort inhibition of rorγt activity and th17 differentiation by a set of novel compounds
topic Research
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4446823/
https://www.ncbi.nlm.nih.gov/pubmed/26021566
http://dx.doi.org/10.1186/s12865-015-0097-9
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