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Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds
BACKGROUND: Retinoic acid receptor-related orphan receptor gamma t (RORγt) is the master regulator of Th17 cell differentiation, which plays a critical role in the pathology of several autoimmune diseases. By directing Th17 cells function, RORγt could be a potential target for drug development for T...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2015
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4446823/ https://www.ncbi.nlm.nih.gov/pubmed/26021566 http://dx.doi.org/10.1186/s12865-015-0097-9 |
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author | Ding, Qingfeng Zhao, Mei Bai, Chuan Yu, Bolan Huang, Zhaofeng |
author_facet | Ding, Qingfeng Zhao, Mei Bai, Chuan Yu, Bolan Huang, Zhaofeng |
author_sort | Ding, Qingfeng |
collection | PubMed |
description | BACKGROUND: Retinoic acid receptor-related orphan receptor gamma t (RORγt) is the master regulator of Th17 cell differentiation, which plays a critical role in the pathology of several autoimmune diseases. By directing Th17 cells function, RORγt could be a potential target for drug development for Th17 related autoimmune disease. METHODS: A Jurkat cell-based reporter assay system was used for screening RORγt inhibitors from a drug-like chemical library, following with mouse Th17 cells differentiation study to identify the effect of targeted compounds in primary T cells. 293T cell-based reporter assay was conducted to determine the cell specificity, and MTT assay was performed to determine the cell toxicity of those compounds. RESULTS: In this study, we identified four lead compounds that suppressed RORγt activity, Th17 differentiation and IL-17A secretion. These candidates displayed inhibition ability on RORγt activity in T cell derived Jurkat cell, but not in 293 T cell, which indicated the restricted effects of these compounds to other cells or tissues. Futhermore, our results demonstrated that these candidates exhibited more robust inhibitory on IL-17 F transcription expression than IL-17A, which is different from one reported compound, SR1001, that mainly suppressed IL-17A, rather than IL-17 F production. CONCLUSIONS: Our study discovered four novel compounds that inhibited RORγt activity and Th17 function, which indicates their potential in therapeutic application of Th17 related autoimmune disorders. |
format | Online Article Text |
id | pubmed-4446823 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2015 |
publisher | BioMed Central |
record_format | MEDLINE/PubMed |
spelling | pubmed-44468232015-05-29 Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds Ding, Qingfeng Zhao, Mei Bai, Chuan Yu, Bolan Huang, Zhaofeng BMC Immunol Research BACKGROUND: Retinoic acid receptor-related orphan receptor gamma t (RORγt) is the master regulator of Th17 cell differentiation, which plays a critical role in the pathology of several autoimmune diseases. By directing Th17 cells function, RORγt could be a potential target for drug development for Th17 related autoimmune disease. METHODS: A Jurkat cell-based reporter assay system was used for screening RORγt inhibitors from a drug-like chemical library, following with mouse Th17 cells differentiation study to identify the effect of targeted compounds in primary T cells. 293T cell-based reporter assay was conducted to determine the cell specificity, and MTT assay was performed to determine the cell toxicity of those compounds. RESULTS: In this study, we identified four lead compounds that suppressed RORγt activity, Th17 differentiation and IL-17A secretion. These candidates displayed inhibition ability on RORγt activity in T cell derived Jurkat cell, but not in 293 T cell, which indicated the restricted effects of these compounds to other cells or tissues. Futhermore, our results demonstrated that these candidates exhibited more robust inhibitory on IL-17 F transcription expression than IL-17A, which is different from one reported compound, SR1001, that mainly suppressed IL-17A, rather than IL-17 F production. CONCLUSIONS: Our study discovered four novel compounds that inhibited RORγt activity and Th17 function, which indicates their potential in therapeutic application of Th17 related autoimmune disorders. BioMed Central 2015-05-29 /pmc/articles/PMC4446823/ /pubmed/26021566 http://dx.doi.org/10.1186/s12865-015-0097-9 Text en © Ding et al.; licensee BioMed Central. 2015 This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated. |
spellingShingle | Research Ding, Qingfeng Zhao, Mei Bai, Chuan Yu, Bolan Huang, Zhaofeng Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds |
title | Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds |
title_full | Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds |
title_fullStr | Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds |
title_full_unstemmed | Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds |
title_short | Inhibition of RORγt activity and Th17 differentiation by a set of novel compounds |
title_sort | inhibition of rorγt activity and th17 differentiation by a set of novel compounds |
topic | Research |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4446823/ https://www.ncbi.nlm.nih.gov/pubmed/26021566 http://dx.doi.org/10.1186/s12865-015-0097-9 |
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