HDAC9 promotes glioblastoma growth via TAZ-mediated EGFR pathway activation

Histone deacetylase 9 (HDAC9), a member of class II HDACs, regulates a wide variety of normal and abnormal physiological functions. We found that HDAC9 is over-expressed in prognostically poor glioblastoma patients. Knockdown HDAC9 decreased proliferation in vitro and tumor formation in vivo. HDAC9...

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Detalles Bibliográficos
Autores principales: Yang, Rui, Wu, Yanan, Wang, Mei, Sun, Zhongfeng, Zou, Jiahua, Zhang, Yundong, Cui, Hongjuan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals LLC 2015
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4480706/
https://www.ncbi.nlm.nih.gov/pubmed/25760078

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4480706/
https://www.ncbi.nlm.nih.gov/pubmed/25760078

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