Effect of N-n-butyl haloperidol iodide on ROS/JNK/Egr-1 signaling in H9c2 cells after hypoxia/reoxygenation

Reactive oxygen species (ROS)-induced oxidative stress in cells is an important pathophysiological process during myocardial ischemia/reperfusion (I/R) injury, and the transcription factor Egr-1 is a master switch for various damage pathways during reperfusion injury. An in vitro model of myocardial...

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Detalles Bibliográficos
Autores principales: Zhang, Yanmei, Liao, Han, Zhong, Shuping, Gao, Fenfei, Chen, Yicun, Huang, Zhanqin, Lu, Shishi, Sun, Ting, Wang, Bin, Li, Weiqiu, Xu, Han, Zheng, Fuchun, Shi, Ganggang
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2015
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4488875/
https://www.ncbi.nlm.nih.gov/pubmed/26134032
http://dx.doi.org/10.1038/srep11809

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4488875/
https://www.ncbi.nlm.nih.gov/pubmed/26134032
http://dx.doi.org/10.1038/srep11809

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