Cargando…
The Hypothermic Influence on CHOP and Ero1-α in an Endoplasmic Reticulum Stress Model of Cerebral Ischemia
Hypoxia induced endoplasmic reticulum stress causes accumulation of unfolded proteins in the endoplasmic reticulum and activates the unfolded protein response, resulting in apoptosis through CCAAT-enhancer-binding protein homologous protein (CHOP) activation. In an in vitro and in vivo model of isch...
Autores principales: | Poone, Gagandip K., Hasseldam, Henrik, Munkholm, Nina, Rasmussen, Rune S., Grønberg, Nina V., Johansen, Flemming F. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4493463/ https://www.ncbi.nlm.nih.gov/pubmed/25989620 http://dx.doi.org/10.3390/brainsci5020178 |
Ejemplares similares
-
Leukocyte infiltration in experimental stroke
por: Grønberg, Nina Vindegaard, et al.
Publicado: (2013) -
Oxidative damage and chemokine production dominate days before immune cell infiltration and EAE disease debut
por: Hasseldam, Henrik, et al.
Publicado: (2016) -
Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia
por: Han, Yu, et al.
Publicado: (2021) -
Geraniol-Mediated Suppression of Endoplasmic Reticulum Stress Protects against Cerebral Ischemia–Reperfusion Injury via the PERK-ATF4-CHOP Pathway
por: Wu, Yu, et al.
Publicado: (2022) -
Ero1-α and PDIs constitute a hierarchical electron transfer network of endoplasmic reticulum oxidoreductases
por: Araki, Kazutaka, et al.
Publicado: (2013)