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rIL-10 enhances IL-10 signalling proteins in foetal alveolar type II cells exposed to hyperoxia
Although the mechanisms by which hyperoxia promotes bronchopulmonary dysplasia are not fully defined, the inability to maintain optimal interleukin (IL)-10 levels in response to injury secondary to hyperoxia seems to play an important role. We previously defined that hyperoxia decreased IL-10 produc...
| Autores principales: | , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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John Wiley & Sons, Ltd
2015
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4511352/ https://www.ncbi.nlm.nih.gov/pubmed/26059905 http://dx.doi.org/10.1111/jcmm.12596 |