Mitochondrial Optic Atrophy (OPA) 1 Processing Is Altered in Response to Neonatal Hypoxic-Ischemic Brain Injury

Perturbation of mitochondrial function and subsequent induction of cell death pathways are key hallmarks in neonatal hypoxic-ischemic (HI) injury, both in animal models and in term infants. Mitoprotective therapies therefore offer a new avenue for intervention for the babies who suffer life-long dis...

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Detalles Bibliográficos
Autores principales: Baburamani, Ana A., Hurling, Chloe, Stolp, Helen, Sobotka, Kristina, Gressens, Pierre, Hagberg, Henrik, Thornton, Claire
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2015
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4613321/
https://www.ncbi.nlm.nih.gov/pubmed/26393574
http://dx.doi.org/10.3390/ijms160922509

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4613321/
https://www.ncbi.nlm.nih.gov/pubmed/26393574
http://dx.doi.org/10.3390/ijms160922509

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