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Mitochondrial Optic Atrophy (OPA) 1 Processing Is Altered in Response to Neonatal Hypoxic-Ischemic Brain Injury
Perturbation of mitochondrial function and subsequent induction of cell death pathways are key hallmarks in neonatal hypoxic-ischemic (HI) injury, both in animal models and in term infants. Mitoprotective therapies therefore offer a new avenue for intervention for the babies who suffer life-long dis...
Autores principales: | Baburamani, Ana A., Hurling, Chloe, Stolp, Helen, Sobotka, Kristina, Gressens, Pierre, Hagberg, Henrik, Thornton, Claire |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2015
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4613321/ https://www.ncbi.nlm.nih.gov/pubmed/26393574 http://dx.doi.org/10.3390/ijms160922509 |
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